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They have also found out I have fibrosis of my bone marrow and are unsure if it's caused by the medication or the CML.

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However, especially in non-aggressive disease (comprising the great majority of patients), at least partial improvement is usually attainable with one regimen or another, and thus the practitioner is obligated to persist with therapeutic trials until no options remain.

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Up to the data cutoff, nine patients died during the study.

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Nevertheless, Gorre and colleagues demonstrated that point mutations in BCR-ABL may be the primary mechanism of acquired resistance to imatinib, suggesting that the tyrosine kinase activity of BCR-ABL remains crucial in advanced disease.

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Click on citations within drug and disease topics in our clinical reference to review the clinical evidence on MEDLINE.

This gene encodes a protein containing a tyrosine kinase activity domain (SH1) in addition to two other regulatory domains (SH2 and SH3) that mediate protein-protein interaction and modulate activation of signal transduction.

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Además, la SG, la supervivencia sin enfermedad, la recidiva y la mortalidad sin recidiva fueron similares entre las dos cohortes.

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Moreover, the results provided herein represent the first mature clinical data to support the longer-term therapeutic promise of potent, intermittent kinase inhibition for the treatment of CML and other human malignancies.

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Because of this prognostic association, a reduction in the BCR-ABL transcript level by 3 log 10 units or more was defined as a major molecular response.

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